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 Brett Finlay PhD
Biotech Lab/Micro/Bioc
Univ of British Columbia
Main Mall The Univ of British Columbia
Vancouver, BC V6T 1Z4
Canada
bfinlay@interchange.ubc.ca
Biosketch:
Dr. B. Brett Finlay is a Professor in
the Biotechnology Laboratory, and the Departments of Biochemistry and Molecular
Biology, and Microbiology and Immunology at the University of British Columbia.
He obtained a B.Sc. (Honors) in Biochemistry at the University of Alberta, where
he also did his Ph.D. (1986) in Biochemistry under Dr. William Paranchych,
studying F-like plasmid conjugation. His post-doctoral studies were performed
with Dr. Stanley Falkow at the Department of Medical Microbiology and Immunology
at Stanford University School of Medicine, where he studied Salmonella invasion
into host cells. In 1989, he joined UBC as an Assistant Professor in the
Biotechnology Laboratory. Dr. Finlay’s research interests are focussed on
host-pathogen interactions, at the molecular level. By combining cell biology
with microbiology, he has been at the forefront of the emerging field called
Cellular Microbiology, making several fundamental discoveries in this field, and
publishing over 200 papers. His laboratory studies several pathogenic bacteria,
with Salmonella and pathogenic E. coli interactions with host cells being the
primary focus. He is well recognized internationally for his work, and has
won several prestigious awards including the 1999 E.W.R. Steacie Prize, the CSM
Fisher Scientific Award, a MRC Scientist, four Howard Hughes International
Research Scholar Awards, a CIHR Distinguished Investigator, BC Biotech
Innovation Award, the Michael Smith Health Research Prize, is a Fellow of the
Royal Society of Canada and the UBC Peter Wall Distinguished Professor. He is a
cofounder of Inimex Pharmaceuticals, Inc., and Director of the SARS Accelerated
Vaccine Initiative. He also serves on several editorial and advisory boards,
and is a strong supporter of communicating science to the public.
Presentation Title:
Salmonella and pathogenic E. coli
interactions with epithelial barrier.
 Back to
Innate Immunity
at the Epithelial Barrier
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